Da har han ikke hørt om de antistoffer som gjør at TSH fortsatt er lavt enda man ikke er hyper.

http://www.suite101.com/article.cfm/gra ... ease/88875 The TSH nightmare
But recent studies show that there is another reason for TSH to be suppressed. Researches in Amsterdam have found that TSH receptor antibodies, even the blocking kind that cause hypothyroidism, may trick the pituitary. The pituitary gland is thought to also have TSH receptors. Besides getting its orders from the hypothalamus, TSH receptors in the pituitary tell it if TSH levels are adequate. Just as TSH receptors located on thyroid cells get tricked by TSH receptor antibodies, TSH receptors in the pituitary gland recognize TSH receptors antibodies as if they were TSH. Thinking there is a high concentration of TSH in the blood (when there are actually high antibody levels and no TSH), the pituitary thinks we’re hyperthyroid and quits secreting TSH.
What a nightmare for thyroid patients. It’s no wonder that after treatment for Graves’ disease, many patients end up complaining of hypothyroid symptoms only to be told that their labs indicate that they’re hyperthyroid. What can be done about this? Insist that your doctor monitor you with levels of thyroid hormone, preferably levels of free T4 and free T3. Also insist on being tested for TSH receptor antibody levels.

Source: LJ Brokken, et al, “Suppression of serum TSH by Graves’Ig: evidence for a functional pituitary TSH receptor,” Journal of Clinical Endocrinology and Metabolism, Oct 2001, 4814-4817.
http://www.hotthyroidology.com/editorial_121.html "Most clinicians will be aware of the fact that TSH levels can remain low, despite clinical euthyroidism and normal concentrations of T4 and T3, in patients treated for Graves’ hyperthyroidism."
http://jcem.endojournals.org/cgi/conten ... /88/9/4135 Thyrotropin Receptor Autoantibodies Are Associated with Continued Thyrotropin Suppression in Treated Euthyroid Graves’ Disease Patients
http://jcem.endojournals.org/cgi/conten ... 86/10/4814 Suppression of Serum TSH by Graves’ Ig: Evidence for a Functional Pituitary TSH Receptor "Antithyroid treatment for Graves’ hyperthyroidism restores euthyroidism clinically within 1–2 months, but it is well known that TSH levels can remain suppressed for many months despite normal free T4 and T3 levels."