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hehe det var nok ikke navnet på rottene...

Her er mer om rottene, fra pdf-en: "Male Wistar rats (Charles-Rivers, Lecco, Italy) weighing 270±300 g were used in this study."

Her fra konklusjonen:

"The results presented in this paper may help to answer another question about the physiological meaning of the effect exerted by 3,5-T2. An increased activity of the c-reducer and c-oxidizer blocks causes the increase in the mitochondrial respiration rate observed in the 3,5-T2 group. In the whole animal, this phenomenon may result in an increased metabolic rate. Because of this, the effect of 3,5-T2 could be important in certain physiological conditions in which rapid energy utilization is required (such as cold exposure, overfeeding, etc.). The effect of 3,5-T2 in improving the cold tolerance of hypothyroid rats supports this hypothesis [19].

3,5-T2, originating from precursor molecules (thyroxine and 3«,3,5-tri-iodo-l-thyronine) through peripheral metabolism, may be a cellular mediator for the short-term effect of thyroid hormones on cellular energetic metabolism, an effect that is independent of protein synthesis [15,27]. The presence of speci®c binding sites for 3,5-T2 in rat liver mitochondria would seem to support this hypothesis [16]."

Kroppen har altså reseptorer for t2. Dat betyr det er et hormon. (som kjent har kroppen ikke reseptorer for t4...)
Her er en opfølgning på det Italienske forsøg fra 1998. Interessant læsning; vist ikke nogen tvivl om at T2 er et vigtigt hormon for energibalancen i vores celler: http://www.ncbi.nlm.nih.gov/pubmed/19878644