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"Is Thyroid Function and Hormone Conversion At the Center of the Mystery?

Dr. John C. Lowe, one of the nation's pioneers in fibromylagia research, has, in a 1997 research study reported in the Clinical Bulletin of Myofascial Therapy, that he found clear relationships between thyroid function and fibromyalgia, and believes that some form of hypometabolism, including thyroid dysfunction, may in part explain FMS.

In studying patients with diagnosed fibromyalgia, Dr. Lowe ran thyroid function tests on each patient. Those with elevated TSH levels (indicating hypothyroidism) did not undergo TRH stimulation tests. Those who had normal TSH levels were subsequently also given a TRH stimulation test.

Of the patients studied:
10.5% had primary hypothyroidism.
36.8% were diagnosed as euthyroid (in normal thyroid state)
52.6% had results, via the TRH test, that were consistent with central hypothyroidism.
Overall, the results of this analysis suggest that approximately 64% of fibromyalgia patients had thyroid hormone deficiencies. And interestingly, the level of primary hypothyroidism found among FMS patients is 10.5 times higher than what you'd expect to find in the general population.

Dr. Lowe cites unpublished studies which indicated that while euthyroid fibromyalgia patients do not have a thyroid hormone deficiency, they benefit as much from receiving T3 as most hypothyroid fibromyalgia patients do from T4. T3 is one of the two thyroid hormones, known commercially as Cytomel, and also a component in the T4/T3 drugs levothyroxine sodium (Synthroid and others), Thyrolar, and the naturally derived Armour Thyroid. Dr. Lowe indicates that a reason for this could be partial cellular resistance to thyroid hormone, as euthyroid fibromyalgia patients resemble patients with thyroid hormone resistance syndromes.

Some HAIT patients who do not resolve their hypothyroidism symptoms while taking T4-only drugs (and having normal levels on TSH tests) have found that taking the T4/T3 combination drugs relieve their hypothyroidism symptoms. This suggests some sort of underlying thyroid hormone resistance syndrome or FMS may also be at play in HAIT patients who do not thrive on the conventional T4 therapies.

Similar linkages between CFS and thyroid hormones have also been suggested. In a 1993 article published in the journal Medical Hypotheses researchers indicated that "...we believe much of the symptomatology [of chronic fatigue syndrome] can be explained by...disturbances of insulin and T3-thyroid hormone functions."

In an article by Raphael Kellman, M.D. in the magazine Alternative Medicine Dr. Kellman indicates that hypothyroidism -- an underactive thyroid -- as well as certain nutritional deficiencies, may underlie many cases of chronic fatigue."

Er jo dette jeg har snakket litt om på denne posten. Antagelig er det en sammenheng med lavt stoffskifte og kronisk utmattelse. Det går jo an å ha begge deler.